Annals of Indian Academy of Neurology
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   2000| January-March  | Volume 3 | Issue 1  
    Online since April 23, 2009

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Multiple Cranial Nerve Involvement In Cryptococcal Meningitis
A Mahadevan, A Kumar, V Santosh, P Satishchandra, S.K Shankar
January-March 2000, 3(1):33-37
Cryptococcal meningitis is an uncommon cause of multiple cranial nerve palsies. This case report illustrates one such case of cryptococcal meningitis clinically manifesting with extensive cranial nerve involvement in an HIV seronegative individual. Histology revealed infiltration of the cranial nerves by cryptococci causing axonal disruption with secondary demyelination in the absence of any evidence of inflammation or vasculitis. We believe that axonal damage underlies the pathogenesis of cranial nerve involvement in cryptococcal meningitis.
[ABSTRACT]   Full text not available  [PDF]
  1,428 166 -
Role Of Acetazolamide In The Treatment Of Partial Epilepsy
T Iype, A Alias, TP Antony, Ravindranathan, M Madusudanan
January-March 2000, 3(1):29-32
This prospective uncontrolled study was done to evaluate the role of acetazolamide as an adjunct in the treatment of partial and secondarily generalized seizures. Fourteen out of 15 patients responded to addition of acetazolamide. The mean follow up was 14.86 months. Three patients subsequently developed tolerance. The only side effect noted was paresthesia of limbs not disabling enough to stop the drug. Acetazolamide is a useful adjunct, in the management of partial and secondarily generalized seizures.
[ABSTRACT]   Full text not available  [PDF]
  1,208 98 -
Paratrigeminal Syndrome Of Raeder
M.A Wani, A.R Kirmani, A.U Ramzan, Wani Haseena
January-March 2000, 3(1):41-42
This 50 year old housewife reported with severe right sided orbital and temporal pain. The clinical examination revealed right third cranial nerve paresis with ptosis and small sized pupil, (Reader's syndrome - Group I). Cerebral angiogrpahy revealed a giant, partially thrombosed aneurysm of the right internal carotid artery. Her facial pain was relieved by carotid ligation. Intracranial internal carotid aneurysms rarely cause paratrigeminal syndrome of Raeder. The mechanism of facial pain and ocular synpathetic involvement is discussed.
[ABSTRACT]   Full text not available  [PDF]
  1,197 106 -
Chemotherapy Induced Hepatitis In Tuberculous Meningitis : A Prospective Study Of 54 Cases
M Tripathi, M.M Wani, M.V Padma, S Jain, M.C Maheshwari
January-March 2000, 3(1):23-28
Fifty four patients of tuberculous meningitis, diagnosed by a standard protocol were followed up for a mean period of 9 months, with periodic assessment of laboratory tests for liver function. A total of 23 patients (42.6%: 10 males and 13 females) developed hepatic dysfunction and 7 of these (30.4%) developed clinical jaundice. Derangement of liver functions was noted in the first month after starting the treatment. Of the various risk factors, a low body mass index and low serum albumin were possible predictors, while presence of increased intracranial tension, previous exposure to anti-tuberculous therapy and presence of extraneural tuberculosis and tuberculous emerged as possible protective factors. Age, sex, basal metabolic rate, concurrent anticonvulsant medication, other laboratory and clinical paremeters were insignificant risk factors.
[ABSTRACT]   Full text not available  [PDF]
  1,163 136 -
Persistent Cerebellar Dysfunction Following Falciparum Malaria
U Seneviratne, R Ganage
January-March 2000, 3(1):39-40
A 49 year old woman presented with one year's history of dysarthria and ataxia. She had 3 attacks of falciparum malaria in the past and developed features of cerebellar dysfunction on the seventh day after the third attack was cured. All three episodes were diagnosed on the basis of positive peripheral blood films for asexual stages of Plasmodium falciparum. The illness reached its peak in two weeks and remained static thereafter. Delayed cerebeller syndrome following falciparum malaria is usually a self-limiting illness with complete recovery (delayed reversible post-malaria cerebellar syndrome). This case illustrates another variety of delayed post-malaria cerebellar syndrome in which cerebellar dysfunction is persistent (delayed persistent post-malaria cerebella syndrome). This could be considered a type of post-malaria neurological syndrome described in the past.
[ABSTRACT]   Full text not available  [PDF]
  1,136 93 -
Neurosciences In India : An Overview
P.N Tandon, M Gourie-Devi
January-March 2000, 3(1):3-21
Full text not available  [PDF]
  785 315 -
Book Reviews
M. Sambasvian
January-March 2000, 3(1):43-44
Full text not available  [PDF]
  749 83 -
Annals Of Indian Academy Of Neurology
M. Gourie-Devi
January-March 2000, 3(1):47-48
Full text not available  [PDF]
  730 62 -
Darab Dastur
January-March 2000, 3(1):45-46
Full text not available  [PDF]
  706 77 -
M. Gourie-Devi
January-March 2000, 3(1):1-1
Full text not available  [PDF]
  625 62 -
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